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            <name>Title</name>
            <description>A name given to the resource</description>
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                <text>Agricultura sostenible</text>
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            <description>An account of the resource</description>
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                <text>Dominio científico: Agricultura sostenible</text>
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          <name>Title</name>
          <description>A name given to the resource</description>
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              <text>Hepatotoxicidad en el paciente crítico: El hígado bajo agresión aguda grave Hepatotoxicity in the critically ill patient: The liver under an acute severe insult</text>
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          <name>Creator</name>
          <description>An entity primarily responsible for making the resource</description>
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              <text>A. García de Lorenzo y Mateos, J. A. Rodríguez Montes</text>
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          <name>Description</name>
          <description>An account of the resource</description>
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              <text>El hígado desempeña un papel fundamental en el metabolismo de la mayoría de los nutrientes pues es un órgano principalmente metabólico y lleva a cabo una serie de procesos fisiológicos y bioquímicos relacionables con el metabolismo proteico y energético. Consideramos al tracto gastrointestinal como una pieza clave en el desarrollo del Fallo o Disfunción Multiorgánica (DMO) como consecuencia de una pérdida de su función de barrera (alteración de la permeabilidad) secundaria a hipoxia frente a toxinas, bioproductos y ocasionalmente bacterias intraluminales, siendo uno de los principales mecanismos fisiopatogénicos el insuficiente flujo de sangre a los órganos esplácnicos. La disfunción hepática y/o alteración en los test de función hepática es un evento común entre los pacientes críticos. Puede estar originada por una cirrosis previa o por causas más inmediatas de insuficiencia- fallo hepático como la sepsis, drogas, transplante hepático o alguna de las múltiples etiologías de la hepatitis.The liver plays an essential role in the metabolism of most of the nutrients since it is a mainly metabolic organ carrying out a series of physiological and metabolic processes related with protein and energy metabolism. The intestinal tract is considered a key element in the development of Multiorgan Dysfunction (MOD) or failure by loosing its barrier function (impaired permeability) against toxins, bio-products and occsaionally intraluminal bacteria secondary to hypoxia, one of the main pathopysiogenic mechanisms being the insufficient blood flow to splacnic organs. Liver dysfunction and/or impairment of liver function test are a common event in critically ill patients. They may be due to previous liver cirrhosis or to more immedite causes of liver failure such as sepsis, drugs, liver transplant or any of the multiple etiologies for hepatitis.</text>
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          <name>Date</name>
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              <text>2008</text>
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          <name>Subject</name>
          <description>The topic of the resource</description>
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              <text>Agreesión, Hígado, Injury, Liver, disfunción, dysfunction</text>
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          <name>Source</name>
          <description>A related resource from which the described resource is derived</description>
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              <text>Nutrición Hospitalaria</text>
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          <name>Publisher</name>
          <description>An entity responsible for making the resource available</description>
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            <elementText elementTextId="144602">
              <text>Arán Ediciones, S. L.</text>
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          <name>Coverage</name>
          <description>The spatial or temporal topic of the resource, the spatial applicability of the resource, or the jurisdiction under which the resource is relevant</description>
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              <text>Nutritional diseases. Deficiency diseases</text>
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          <name>Relation</name>
          <description>A related resource</description>
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            <elementText elementTextId="144604">
              <text>&lt;a href="http://scielo.isciii.es/scielo.php?script=sci_arttext&amp;amp;pid=S0212-16112008000600004" target="_blank" rel="noreferrer noopener"&gt;http://scielo.isciii.es/scielo.php?script=sci_arttext&amp;amp;pid=S0212-16112008000600004&lt;/a&gt;</text>
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